myocardial infarction
myocardial ischemia
myocardial damage
acute myocardial infarction
Objective To study the effect of measle virus on myocardial enzymogram.
Objective: To investigate the effect of intramyocardial bone marrow cell injection on myocardial perfusion and LV function in patients with chronic myocardial ischemia.
The epicardial electrogram was used to measure the myocardial ischemia degree.
Perform in the clinic if no bruits, ventricular tachycardia, recent stroke, or myocardial infarction.
Myocardial adenosine triphosphate (ATP) and phosphocreatine (CrP) were measured with 31P magnetic resonance.
The antibody detection may serve as a diagnostic index for VMC and DCM.②The specific antigen is probably human myocardial adenine nucleotide translocator.
Abstract: Objective:to make observation on the effect of qi-boosting &yin-nourishing formula on myocardial damage Method:we adopted the model of myocardial damage caused by amycin.
The main aim of the study was to analyze their specific complications, including heart arrhythmia, pulmonary infection, hepatic coma, alienism, myocardial infaction and diabetic ketoacidosis.
Objective:To evaluate the myocardial protective effects of HTK solution on immature and cyanosed myocardium in cardiac operation,through comparison with ST.
Mismatches between myocardial fatty acid uptake and utilization lead to the accumulation of cardiotoxic lipid species,and cause lipotoxic cardiomyopathy.
Objective: To understand relation between the clockwise horizontal plane T loop in vectorcardiogram (VCG) and myocardial ischemia.
Objective:To discuss incidence,clinical significance and result of bradycardic dysfunction of sinoatrial node in inferior wall acute myocardial infarction(AMI).
Association between the -2518G/A polymorphism in the monocyte chemoattractant protein-1 (MCP-1) gene and myocardial infarction in Tunisian patients.
They play protective roles in cytoprotection in myocardial ischemia reperfusion injury,systemic inflammatory response syndrome and apoptosis,and have more advantages in cytoprotection than drugs.
Abstract: Phospholamban (PLB) is a prominent regulator of myocardial contractility and a reversible inhibitor of the cardiac sarcoplasmic reticulum Ca2+ ATPase activity.
Expressions of S-adenosylmethionine synthetase gene, arginase gene and oxidant stress related genes in myocardial tissue of normal and diabetic rats were assayed by DNA microarray.
Conclusions: The myocardial bridge is an anatomic structure with some functions, such as supporting and fixing aeteria coronaria, increasing arteriotony and raising blood supply of myocardium.
The influence of the drug on the cytopathy of the myocardial cells,its effects on virus replication and the content of troponin in the supernatants of the cultured cells were kept under observation.
We recorded the changes of the heart rate the blood pressure, and the left ventricle pressure, and observed Ginkgetin effect on reducing the myocardial infarct death rate of AMI - reperfused rabbits.
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