peptidoleukotriene release
肽类白三烯释放
peptidoleukotriene synthesis
肽类白三烯合成
peptidoleukotriene inhibitor
肽类白三烯抑制剂
peptidoleukotriene receptor
肽类白三烯受体
peptidoleukotriene levels
肽类白三烯水平
peptidoleukotriene production
肽类白三烯生成
peptidoleukotriene pathway
肽类白三烯通路
peptidoleukotriene antagonist
肽类白三烯拮抗剂
peptidoleukotriene activity
肽类白三烯活性
peptidoleukotriene formation
肽类白三烯形成
the release of peptidoleukotriene contributes significantly to bronchoconstriction in asthmatic patients.
肽类白三烯的释放在哮喘患者的支气管收缩中起着重要作用。
researchers observed that peptidoleukotriene production increases during allergic reactions.
研究人员观察到，在过敏反应期间肽类白三烯的生成会增加。
antagonists blocking the peptidoleukotriene receptor are effective treatments for allergic rhinitis.
阻断肽类白三烯受体的拮抗剂是治疗过敏性鼻炎的有效疗法。
biosynthesis of peptidoleukotriene depends on the 5-lipoxygenase pathway in inflammatory cells.
肽类白三烯的生物合成依赖于炎症细胞中的5-脂氧合酶途径。
increased peptidoleukotriene levels were detected in the sputum of chronic bronchitis sufferers.
在慢性支气管炎患者的痰液中检测到肽类白三烯水平升高。
pharmacological inhibition of peptidoleukotriene action reduces airway inflammation significantly.
药理学抑制肽类白三烯的作用可显著减少气道炎症。
ltc4 is a major peptidoleukotriene involved in the pathogenesis of asthma.
LTC4是参与哮喘发病机制的主要肽类白三烯。
the study examined peptidoleukotriene metabolism in patients with aspirin-exacerbated respiratory disease.
该研究检查了阿司匹林加重型呼吸道疾病患者中的肽类白三烯代谢。
specific assays are required to accurately measure peptidoleukotriene concentration in blood samples.
需要特定的检测方法才能准确测量血液样本中的肽类白三烯浓度。
scientists are investigating the role of peptidoleukotriene signaling in pulmonary fibrosis.
科学家正在研究肽类白三烯信号在肺纤维化中的作用。
mast cells are a primary source of peptidoleukotriene release during anaphylaxis.
在过敏性休克期间，肥大细胞是肽类白三烯释放的主要来源。
genetic variations can affect peptidoleukotriene receptor sensitivity among different individuals.
基因变异可能影响不同个体对肽类白三烯受体的敏感性。
peptidoleukotriene release
肽类白三烯释放
peptidoleukotriene synthesis
肽类白三烯合成
peptidoleukotriene inhibitor
肽类白三烯抑制剂
peptidoleukotriene receptor
肽类白三烯受体
peptidoleukotriene levels
肽类白三烯水平
peptidoleukotriene production
肽类白三烯生成
peptidoleukotriene pathway
肽类白三烯通路
peptidoleukotriene antagonist
肽类白三烯拮抗剂
peptidoleukotriene activity
肽类白三烯活性
peptidoleukotriene formation
肽类白三烯形成
the release of peptidoleukotriene contributes significantly to bronchoconstriction in asthmatic patients.
肽类白三烯的释放在哮喘患者的支气管收缩中起着重要作用。
researchers observed that peptidoleukotriene production increases during allergic reactions.
研究人员观察到，在过敏反应期间肽类白三烯的生成会增加。
antagonists blocking the peptidoleukotriene receptor are effective treatments for allergic rhinitis.
阻断肽类白三烯受体的拮抗剂是治疗过敏性鼻炎的有效疗法。
biosynthesis of peptidoleukotriene depends on the 5-lipoxygenase pathway in inflammatory cells.
肽类白三烯的生物合成依赖于炎症细胞中的5-脂氧合酶途径。
increased peptidoleukotriene levels were detected in the sputum of chronic bronchitis sufferers.
在慢性支气管炎患者的痰液中检测到肽类白三烯水平升高。
pharmacological inhibition of peptidoleukotriene action reduces airway inflammation significantly.
药理学抑制肽类白三烯的作用可显著减少气道炎症。
ltc4 is a major peptidoleukotriene involved in the pathogenesis of asthma.
LTC4是参与哮喘发病机制的主要肽类白三烯。
the study examined peptidoleukotriene metabolism in patients with aspirin-exacerbated respiratory disease.
该研究检查了阿司匹林加重型呼吸道疾病患者中的肽类白三烯代谢。
specific assays are required to accurately measure peptidoleukotriene concentration in blood samples.
需要特定的检测方法才能准确测量血液样本中的肽类白三烯浓度。
scientists are investigating the role of peptidoleukotriene signaling in pulmonary fibrosis.
科学家正在研究肽类白三烯信号在肺纤维化中的作用。
mast cells are a primary source of peptidoleukotriene release during anaphylaxis.
在过敏性休克期间，肥大细胞是肽类白三烯释放的主要来源。
genetic variations can affect peptidoleukotriene receptor sensitivity among different individuals.
基因变异可能影响不同个体对肽类白三烯受体的敏感性。
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