cholangiocyte damage
膽管上皮細胞損傷
cholangiocyte proliferation
膽管上皮細胞增殖
cholangiocyte apoptosis
膽管上皮細胞凋亡
cholangiocyte dysfunction
膽管上皮細胞功能障礙
cholangiocyte injury
膽管上皮細胞損傷
cholangiocyte activation
膽管上皮細胞活化
cholangiocyte secretion
膽管上皮細胞分泌
cholangiocyte isolation
膽管上皮細胞分離
cholangiocyte culture
膽管上皮細胞培養
cholangiocyte phenotype
膽管上皮細胞表型
chronic cholangiocyte injury leads to progressive fibrosis in the bile ducts.
慢性膽管上皮細胞損傷會導致膽管進行性纖維化。
cholangiocyte proliferation is a key feature of many cholestatic liver diseases.
膽管上皮細胞增殖是許多膽汁淤積性肝病的關鍵特徵。
researchers are studying cholangiocyte apoptosis to understand the mechanisms of biliary atresia.
研究人員正在研究膽管上皮細胞凋亡以了解膽道閉鎖的機制。
bile acids can induce cholangiocyte senescence through oxidative stress pathways.
膽汁酸可通過氧化應激途徑誘導膽管上皮細胞衰老。
the integrity of cholangiocyte tight junctions is essential for bile formation.
膽管上皮細胞緊密連接的完整性對膽汁生成至關重要。
cholangiocyte-derived cytokines play a crucial role in hepatic inflammation.
由膽管上皮細胞產生的細胞因子在肝臟炎症中發揮關鍵作用。
impaired cholangiocyte secretion contributes to the pathogenesis of cholestasis.
膽管上皮細胞分泌功能受損會促進膽汁淤積的發病機制。
cholangiocyte metabolism of bile acids is altered in patients with primary sclerosing cholangitis.
原發性硬化性膽管炎患者膽管上皮細胞對膽汁酸的代謝發生改變。
the study revealed that cholangiocyte signaling pathways are dysregulated during cholestasis.
該研究顯示,在膽汁淤積期間,膽管上皮細胞的信號傳導途徑出現異常。
cholangiocyte regeneration capacity declines with age and in chronic liver disease.
隨著年齡增長和慢性肝病的發展,膽管上皮細胞的再生能力下降。
targeting cholangiocyte activation may offer new therapeutic approaches for liver fibrosis.
針對膽管上皮細胞的活化可能為肝纖維化的治療提供新方法。
cholangiocyte death triggers an inflammatory response that promotes hepatic fibrosis.
膽管上皮細胞死亡會觸發促進肝纖維化的炎症反應。
genetic mutations affecting cholangiocyte function have been identified in several liver disorders.
已在多種肝臟疾病中發現影響膽管上皮細胞功能的基因突變。
cholangiocyte transdifferentiation is a critical process in the development of cholangiocarcinoma.
膽管上皮細胞的轉分化是膽管癌發展中的關鍵過程。
understanding cholangiocyte transport mechanisms is essential for developing new treatments.
了解膽管上皮細胞的運輸機制對於開發新療法至關重要。
cholangiocyte damage
膽管上皮細胞損傷
cholangiocyte proliferation
膽管上皮細胞增殖
cholangiocyte apoptosis
膽管上皮細胞凋亡
cholangiocyte dysfunction
膽管上皮細胞功能障礙
cholangiocyte injury
膽管上皮細胞損傷
cholangiocyte activation
膽管上皮細胞活化
cholangiocyte secretion
膽管上皮細胞分泌
cholangiocyte isolation
膽管上皮細胞分離
cholangiocyte culture
膽管上皮細胞培養
cholangiocyte phenotype
膽管上皮細胞表型
chronic cholangiocyte injury leads to progressive fibrosis in the bile ducts.
慢性膽管上皮細胞損傷會導致膽管進行性纖維化。
cholangiocyte proliferation is a key feature of many cholestatic liver diseases.
膽管上皮細胞增殖是許多膽汁淤積性肝病的關鍵特徵。
researchers are studying cholangiocyte apoptosis to understand the mechanisms of biliary atresia.
研究人員正在研究膽管上皮細胞凋亡以了解膽道閉鎖的機制。
bile acids can induce cholangiocyte senescence through oxidative stress pathways.
膽汁酸可通過氧化應激途徑誘導膽管上皮細胞衰老。
the integrity of cholangiocyte tight junctions is essential for bile formation.
膽管上皮細胞緊密連接的完整性對膽汁生成至關重要。
cholangiocyte-derived cytokines play a crucial role in hepatic inflammation.
由膽管上皮細胞產生的細胞因子在肝臟炎症中發揮關鍵作用。
impaired cholangiocyte secretion contributes to the pathogenesis of cholestasis.
膽管上皮細胞分泌功能受損會促進膽汁淤積的發病機制。
cholangiocyte metabolism of bile acids is altered in patients with primary sclerosing cholangitis.
原發性硬化性膽管炎患者膽管上皮細胞對膽汁酸的代謝發生改變。
the study revealed that cholangiocyte signaling pathways are dysregulated during cholestasis.
該研究顯示,在膽汁淤積期間,膽管上皮細胞的信號傳導途徑出現異常。
cholangiocyte regeneration capacity declines with age and in chronic liver disease.
隨著年齡增長和慢性肝病的發展,膽管上皮細胞的再生能力下降。
targeting cholangiocyte activation may offer new therapeutic approaches for liver fibrosis.
針對膽管上皮細胞的活化可能為肝纖維化的治療提供新方法。
cholangiocyte death triggers an inflammatory response that promotes hepatic fibrosis.
膽管上皮細胞死亡會觸發促進肝纖維化的炎症反應。
genetic mutations affecting cholangiocyte function have been identified in several liver disorders.
已在多種肝臟疾病中發現影響膽管上皮細胞功能的基因突變。
cholangiocyte transdifferentiation is a critical process in the development of cholangiocarcinoma.
膽管上皮細胞的轉分化是膽管癌發展中的關鍵過程。
understanding cholangiocyte transport mechanisms is essential for developing new treatments.
了解膽管上皮細胞的運輸機制對於開發新療法至關重要。
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