marcks protein plays a crucial role in signal transduction pathways within eukaryotic cells.
Marcks蛋白在真核細胞的訊號傳導途徑中扮演關鍵角色。
the phosphorylation of marcks regulates its interaction with actin filaments and modulates cell motility.
Marcks的磷酸化調節其與肌動蛋白絲的交互作用，並調控細胞運動性。
researchers have identified marcks as a key substrate for protein kinase c in various cell types.
研究人員已將Marcks識別為各種細胞類型中蛋白激酶C的關鍵底物。
marcks expression is significantly upregulated during neuronal development and synaptic plasticity.
在神經元發育和突觸可塑性期間，Marcks的表達會顯著上調。
mutations in the marcks gene have been associated with neurodevelopmental disorders in clinical studies.
臨床研究顯示，Marcks基因的突變與神經發育障礙有關。
the marcks protein contains multiple its sub phosphorylation sites that controlcellular localization.
Marcks蛋白含有多個亞單位磷酸化位點，這些位點控制細胞定位。
inhibiting marcks function through pharmacological approaches affects cell migration and invasion.
通過藥理學方法抑制Marcks功能會影響細胞遷移和侵襲。
marcks is targeted to the plasma membrane through n-terminal myristoylation and electrostatic interactions.
Marcks通過N端肉豆蔻醯化和靜電相互作用被靶向至質膜。
calcium influx triggers rapid phosphorylation of marcks by downstream kinases.
鈣離子流入會觸發下游激酶對Marcks的快速磷酸化。
marcks interacts with calmodulin in a calcium-dependent manner, regulating various cellular processes.
Marcks以鈣離子依賴的方式與鈣調蛋白相互作用，調控各種細胞過程。
the knockdown of marcks using sirna results in dramatic changes to cell morphology and adhesion.
使用siRNA敲低Marcks會導致細胞形態和黏附性的顯著變化。
marcks serves as a molecular hub integrating signals from multiple signaling pathways.
Marcks作為一個分子樞紐，整合來自多個訊號途徑的訊號。
marcks protein plays a crucial role in signal transduction pathways within eukaryotic cells.
Marcks蛋白在真核細胞的訊號傳導途徑中扮演關鍵角色。
the phosphorylation of marcks regulates its interaction with actin filaments and modulates cell motility.
Marcks的磷酸化調節其與肌動蛋白絲的交互作用，並調控細胞運動性。
researchers have identified marcks as a key substrate for protein kinase c in various cell types.
研究人員已將Marcks識別為各種細胞類型中蛋白激酶C的關鍵底物。
marcks expression is significantly upregulated during neuronal development and synaptic plasticity.
在神經元發育和突觸可塑性期間，Marcks的表達會顯著上調。
mutations in the marcks gene have been associated with neurodevelopmental disorders in clinical studies.
臨床研究顯示，Marcks基因的突變與神經發育障礙有關。
the marcks protein contains multiple its sub phosphorylation sites that controlcellular localization.
Marcks蛋白含有多個亞單位磷酸化位點，這些位點控制細胞定位。
inhibiting marcks function through pharmacological approaches affects cell migration and invasion.
通過藥理學方法抑制Marcks功能會影響細胞遷移和侵襲。
marcks is targeted to the plasma membrane through n-terminal myristoylation and electrostatic interactions.
Marcks通過N端肉豆蔻醯化和靜電相互作用被靶向至質膜。
calcium influx triggers rapid phosphorylation of marcks by downstream kinases.
鈣離子流入會觸發下游激酶對Marcks的快速磷酸化。
marcks interacts with calmodulin in a calcium-dependent manner, regulating various cellular processes.
Marcks以鈣離子依賴的方式與鈣調蛋白相互作用，調控各種細胞過程。
the knockdown of marcks using sirna results in dramatic changes to cell morphology and adhesion.
使用siRNA敲低Marcks會導致細胞形態和黏附性的顯著變化。
marcks serves as a molecular hub integrating signals from multiple signaling pathways.
Marcks作為一個分子樞紐，整合來自多個訊號途徑的訊號。
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