overperfusion syndrome
過灌注綜合症
cerebral overperfusion
腦過灌注
overperfusion injury
過灌注損傷
tissue overperfusion
組織過灌注
pulmonary overperfusion
肺過灌注
prevent overperfusion
預防過灌注
renal overperfusion
腎過灌注
overperfusion causes
過灌注原因
overperfusion of
過灌注的
treating overperfusion
治療過灌注
cerebral overperfusion syndrome can occur after carotid endarterectomy in patients with compromised autoregulation.
在自主调节功能受损的患者中，頸動脈內膜剝除術後可能發生腦過灌注綜合徵。
tissue overperfusion leads to edema and cellular damage due to excessive oxygen delivery beyond metabolic needs.
組織過灌注會導致水腫和細胞損傷，這是因為氧氣供應過多超過了代謝需求。
the physician monitored for signs of organ overperfusion during the aggressive fluid resuscitation protocol.
醫生在積極的體液復甦方案中監測器官過灌注的跡象。
capillary overperfusion causes increased hydrostatic pressure that promotes plasma extravasation into interstitial spaces.
毛細血管過灌注會導致靜水壓升高，促進血漿滲出到間質空間。
regional overperfusion may explain the preferential distribution of contrast enhancement in certain vascular territories.
區域性過灌注可能解釋了某些血管區域對比劑增強的優先分布。
overperfusion injury to the brainstem resulted from the rapid correction of chronic cerebral hypoperfusion.
腦幹的過灌注損傷是由於迅速糾正慢性腦灌注不足所致。
the cardiology team adjusted the inotropic support to prevent myocardial overperfusion and subsequent reperfusion injury.
心臟科團隊調整了正性肌力支持，以防止心肌過灌注和後續的再灌注損傷。
post-operative monitoring revealed pulmonary overperfusion that contributed to the development of pulmonary edema.
術後監測發現肺過灌注，這促成了肺水腫的發展。
hepatic overperfusion syndrome was observed following successful revascularization of the previously ischemic liver.
在成功恢復 previously 灌注的缺血肝臟後，觀察到肝過灌注綜合徵。
the patient developed renal overperfusion after aggressive volume expansion, necessitating diuretic therapy.
患者在接受積極的體積擴張後出現腎過灌注，需要利尿治療。
risk factors for cerebral overperfusion include severe carotid stenosis and inadequate collateral circulation.
腦過灌注的風險因素包括嚴重的頸動脈狹窄和不充分的側支循環。
pulmonary overperfusion can occur in conditions where left ventricular output suddenly increases.
在左心室輸出突然增加的情況下，可能會發生肺過灌注。
overperfusion syndrome
過灌注綜合症
cerebral overperfusion
腦過灌注
overperfusion injury
過灌注損傷
tissue overperfusion
組織過灌注
pulmonary overperfusion
肺過灌注
prevent overperfusion
預防過灌注
renal overperfusion
腎過灌注
overperfusion causes
過灌注原因
overperfusion of
過灌注的
treating overperfusion
治療過灌注
cerebral overperfusion syndrome can occur after carotid endarterectomy in patients with compromised autoregulation.
在自主调节功能受损的患者中，頸動脈內膜剝除術後可能發生腦過灌注綜合徵。
tissue overperfusion leads to edema and cellular damage due to excessive oxygen delivery beyond metabolic needs.
組織過灌注會導致水腫和細胞損傷，這是因為氧氣供應過多超過了代謝需求。
the physician monitored for signs of organ overperfusion during the aggressive fluid resuscitation protocol.
醫生在積極的體液復甦方案中監測器官過灌注的跡象。
capillary overperfusion causes increased hydrostatic pressure that promotes plasma extravasation into interstitial spaces.
毛細血管過灌注會導致靜水壓升高，促進血漿滲出到間質空間。
regional overperfusion may explain the preferential distribution of contrast enhancement in certain vascular territories.
區域性過灌注可能解釋了某些血管區域對比劑增強的優先分布。
overperfusion injury to the brainstem resulted from the rapid correction of chronic cerebral hypoperfusion.
腦幹的過灌注損傷是由於迅速糾正慢性腦灌注不足所致。
the cardiology team adjusted the inotropic support to prevent myocardial overperfusion and subsequent reperfusion injury.
心臟科團隊調整了正性肌力支持，以防止心肌過灌注和後續的再灌注損傷。
post-operative monitoring revealed pulmonary overperfusion that contributed to the development of pulmonary edema.
術後監測發現肺過灌注，這促成了肺水腫的發展。
hepatic overperfusion syndrome was observed following successful revascularization of the previously ischemic liver.
在成功恢復 previously 灌注的缺血肝臟後，觀察到肝過灌注綜合徵。
the patient developed renal overperfusion after aggressive volume expansion, necessitating diuretic therapy.
患者在接受積極的體積擴張後出現腎過灌注，需要利尿治療。
risk factors for cerebral overperfusion include severe carotid stenosis and inadequate collateral circulation.
腦過灌注的風險因素包括嚴重的頸動脈狹窄和不充分的側支循環。
pulmonary overperfusion can occur in conditions where left ventricular output suddenly increases.
在左心室輸出突然增加的情況下，可能會發生肺過灌注。
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