peptidoleukotrienes cause
肽類白三烯引起
peptidoleukotriene antagonist
肽類白三烯拮抗劑
peptidoleukotriene receptor
肽類白三烯受體
peptidoleukotrienes inhibit
肽類白三烯抑制
peptidoleukotriene synthesis
肽類白三烯合成
peptidoleukotriene release
肽類白三烯釋放
peptidoleukotriene levels
肽類白三烯水平
peptidoleukotriene production
肽類白三烯生成
peptidoleukotriene pathway
肽類白三烯途徑
peptidoleukotriene activity
肽類白三烯活性
peptidoleukotrienes are potent bronchoconstrictors involved in the pathogenesis of asthma.
肽類白三烯是強效的支氣管收縮劑，參與哮喘的發病機制。
antagonists of peptidoleukotrienes are effective in treating allergic rhinitis symptoms.
肽類白三烯拮抗劑在治療過敏性鼻炎症狀方面是有效的。
researchers measured the release of peptidoleukotrienes during the hypersensitivity reaction.
研究人員測量了過敏反應期間肽類白三烯的釋放。
cysteinyl peptidoleukotrienes cause edema by increasing vascular permeability in tissues.
半胱氨醯肽類白三烯通過增加組織中的血管通透性引起水腫。
montelukast functions by blocking specific peptidoleukotriene receptors in the airways.
孟魯司特通過阻斷氣道中特定的肽類白三烯受體發揮作用。
biosynthesis of peptidoleukotrienes depends on the 5-lipoxygenase enzyme pathway.
肽類白三烯的生物合成依賴於5-脂氧合酶酶途徑。
high levels of peptidoleukotrienes were detected in the patients' urine samples.
在患者的尿液樣本中檢測到高水平的肽類白三烯。
inhibiting peptidoleukotriene production can significantly reduce airway inflammation.
抑制肽類白三烯的產生可以顯著減少氣道炎症。
the biological activity of peptidoleukotrienes was studied using isolated cells.
使用分離的細胞研究了肽類白三烯的生物活性。
peptidoleukotrienes play a critical role in anaphylactic shock and hypotension.
肽類白三烯在過敏性休克和低血壓中起關鍵作用。
doctors prescribe peptidoleukotriene receptor antagonists for chronic asthma management.
醫生開具肽類白三烯受體拮抗劑用於慢性哮喘的管理。
peptidoleukotrienes cause
肽類白三烯引起
peptidoleukotriene antagonist
肽類白三烯拮抗劑
peptidoleukotriene receptor
肽類白三烯受體
peptidoleukotrienes inhibit
肽類白三烯抑制
peptidoleukotriene synthesis
肽類白三烯合成
peptidoleukotriene release
肽類白三烯釋放
peptidoleukotriene levels
肽類白三烯水平
peptidoleukotriene production
肽類白三烯生成
peptidoleukotriene pathway
肽類白三烯途徑
peptidoleukotriene activity
肽類白三烯活性
peptidoleukotrienes are potent bronchoconstrictors involved in the pathogenesis of asthma.
肽類白三烯是強效的支氣管收縮劑，參與哮喘的發病機制。
antagonists of peptidoleukotrienes are effective in treating allergic rhinitis symptoms.
肽類白三烯拮抗劑在治療過敏性鼻炎症狀方面是有效的。
researchers measured the release of peptidoleukotrienes during the hypersensitivity reaction.
研究人員測量了過敏反應期間肽類白三烯的釋放。
cysteinyl peptidoleukotrienes cause edema by increasing vascular permeability in tissues.
半胱氨醯肽類白三烯通過增加組織中的血管通透性引起水腫。
montelukast functions by blocking specific peptidoleukotriene receptors in the airways.
孟魯司特通過阻斷氣道中特定的肽類白三烯受體發揮作用。
biosynthesis of peptidoleukotrienes depends on the 5-lipoxygenase enzyme pathway.
肽類白三烯的生物合成依賴於5-脂氧合酶酶途徑。
high levels of peptidoleukotrienes were detected in the patients' urine samples.
在患者的尿液樣本中檢測到高水平的肽類白三烯。
inhibiting peptidoleukotriene production can significantly reduce airway inflammation.
抑制肽類白三烯的產生可以顯著減少氣道炎症。
the biological activity of peptidoleukotrienes was studied using isolated cells.
使用分離的細胞研究了肽類白三烯的生物活性。
peptidoleukotrienes play a critical role in anaphylactic shock and hypotension.
肽類白三烯在過敏性休克和低血壓中起關鍵作用。
doctors prescribe peptidoleukotriene receptor antagonists for chronic asthma management.
醫生開具肽類白三烯受體拮抗劑用於慢性哮喘的管理。
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